Politzer's text-book of the diseases of the ear and adjacent organs : for students and practitioners / translated by Oscar Dodd ; edited by Sir William Dalby.

  • Politzer, Adam, 1835-1920.
Date:
1894
Catalogue details

Licence: Public Domain Mark

Credit: Politzer's text-book of the diseases of the ear and adjacent organs : for students and practitioners / translated by Oscar Dodd ; edited by Sir William Dalby. Source: Wellcome Collection.

    642/760 (page 626)
    The treatment of anaemia of the internal ear depends on its cause. The aural symptoms after great loss of blood require no local treat- ment, as they recede with the disappearance of the general anaemia. In the angioneurotic forms the internal use of bromide of potassium and of quinine, and galvanization of the sympathetic in the throat, are recommended. In chronic general anaemia, besides strengthen- ing diet and residence in Alpine air, the internal use of easily-digested iron remedies, such as mineral waters containing iron, is to be advised, and when circumstances permit of it, a course of iron baths is of good service. 3. Hemorrhage into the Labyrinth. Small extravasations in the labyrinth occur as a consequence of severe and rapidly-developed hyperaemia most frequently in the course of typhus, variola, scarlet fever, diphtheria (Moos), mumps (Toynbee), nephritis, diabetes (Steinbrügge), leucocythaemia, per- nicious anaemia, and upon death from suffocation with or without simultaneous ecchymoses on the mucous membrane of the middle ear. The rupture of the bloodvessels is favoured by atheromatous degeneration of the arteries and by chronic congestive hyperaemia from disease of the heart. The sites of the ecchymoses are most frequently the lamina spiralis and the external surface of the modiolus, more rarely the saccule of the vestibule and the ampullae. The extravasated blood may be absorbed, or the blood-colouring matter may remain as granular or flaky pigment. Larger effusions of blood into the labyrinth result: (1) From fractures of the skull and fissures of the petrous bone. In one case observed by me (A. f. 0., vol. ii.), in which a fissure proceeding from the back of the head passed through both petrous bones, total deafness set in with symptoms of the apoplectic form of Meniere's disease, in consequence of a great effusion of blood into the cavity of the labyrinth. Lucae observed after fissure of the petrous bone simultaneous effusion of blood into the labyrinth and into the tympanic cavity. (2) From severe concussion of the skull, as is proved by a case of gunshot-fracture of the mastoid process observed by Moos (A. f. A. u. 0., vol. ii.), in which without lesion of the labyrinth an effusion of blood was found in it. (3) From extensive carious processes in the temporal bone, particularly from caries and necrosis of the pyramid of the petrous bone (Knapp). In several such cases, in which the capsule of the labyrinth was not injured, I found a few sections of the labyrinth, especially the cochlea,