An annotated catalogue of the mutant genes of the house mouse / H. Grüneberg.

  • Grüneberg, H. (Hans)
Date:
1956
Catalogue details

Licence: Attribution-NonCommercial 4.0 International (CC BY-NC 4.0)

Credit: An annotated catalogue of the mutant genes of the house mouse / H. Grüneberg. Source: Wellcome Collection.

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    references 27 Grüneberg, H. (1956b). Hereditary lesions of the labyrinth in the mouse. Brit, med: Bull, 12, 153-7; (for Twirler and Zigzag, discovered but not yet described by Dr. Mary F. Lyon). Hauschka, T. S. and Brown, E. (1954). Influence of maternal age on penetrance of eyelids open in newborn mice. Ann. N.Y. Acad. Sci., 57, 465-75. Hertwig, p. (1951). Entwicklungsgeschichtliche Untersuchungen über Bewegungsstörungen bei Mäusen. Verh. anat. Ges., Jena, 97-107. Hertwig, P. (1955). Der Hydrops-'&izmm. Vielfache Auswirkungen einer durch Röntgenbestrahlung von Spermatogonien entstandenen Erbänderung bei der Hausmaus. Züchter, 25, 194-8. tîESTON, W. E. (1951). The vestigial tail mouse, a new recessive mutation.. J. Hered., 42, 11^. HoECKER, G., Martinez, A., Markovic, S. and Pizzaro, O. (1954). Agitans, a mutation with neurological effects in the house mouse. /. Hered., 45,10-14. Hollander, W. F. and Strong, L. C. (1951). Pintail, a dominant mutation linked with brown in the house mouse. J. Hered., 42, 179-82. Holman, S. p. (1951). The hook-tailed mouse, a mutation in the house mouse aff'ecting the tail and the anus. J. Hered., 42, 305-6. Ingalls, a. M., Dickie, M. M. and Snell, G. D. (1950). Obese, a new rnutation in the house mouse. J. Hered., 41, 317-18. Karli, p. (1951). Rétines sans cellules visuelles. Récherches morphologiques, physiologiques et physiopathologiques chez les rongeurs. Arch. Anat., Strasbourg, 35, 1-75. King, J. W. B. (1955). Observations on the mutant 'pygmy' in the house mouse. /. Genet., 53, 487-97. Kunze, H. G. (1954). Die Erythropoese bei einer erblichen Anämie rönt- genmutierter Mäuse. Folia haemat., Lpz., 72, 391-436. Landauer, W. (1952). Brachypodism, a recessive mutation of house-mice. J. Hered., 43, 293-8. Lyon, M. F. (1953). Absence of otoliths in the mouse: an effect of the pallid mutant. J. Genet., 51, 638-50. Lyon, M. F. (1955a). The developmental origin of hereditary absence of otoliths in mice. J. Embryol. exp. Morph., 3, 230-41. Lyon, M. F. (1955b). Ataxia—a new recessive mutant of the house mouse. /. Hered., 46, 77-80. McNutt, W. (1954). Gene expression as influenced by genetic background as illustrated by ventral tubercles on cervical vertebrae in mice. J. Hered., 45, 235-40. Martinez, A, and Sirlin, J. L. (1955). Neurohistology of the agitans mouse. J. сотр. Neurol., 103, 131-7. Mayer, J. (1953). Genetic, traumatic and environmental factors in the etiology of obesity. Physiol. Rev., 33, 472-508. Mayer, J., Russell, R. E., Bates, M. W. and Dickie, M. M. (1953). Metabolic, nutritional and endocrine studies of the hereditary obesity-diabetes mellitus syndrome in mice and mechanism of its development. Metabolism, 2, 9-21. Michelson, a. M., Russell, E. S. and Harman, P. J. (1955). Dystrophia muscularis : a hereditary primary myopathy in the house mouse. Proc. nat. Acad. Sci., Wash., 41, 1079-84. Miller, D. S. and Potas, M. Z. (1955). Cordovan, a new allele of black and brown color in the mouse. J. Hered., 46, 293-6.