Interstitial gingivitis and pyorrhoea alveolaris.

  • Talbot, Eugene S. (Eugene Solomon), 1847-1924.
Date:
1913
Catalogue details

Licence: Public Domain Mark

Credit: Interstitial gingivitis and pyorrhoea alveolaris. Source: Wellcome Collection.

Provider: This material has been provided by the Augustus C. Long Health Sciences Library at Columbia University and Columbia University Libraries/Information Services, through the Medical Heritage Library. The original may be consulted at the the Augustus C. Long Health Sciences Library at Columbia University and Columbia University.

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    extent, the profession has come to realize the importance of the two periods and the teaching of the pathology and bacteriology of the disease is much simplified and better understood. The great law of medical science, that to know the cause is half the treatment, is as applicable to diseases of the mouth as to any other specialty of medicine, and is as applicable to dental problems as to those of biology generally. Treatment of any disease without knowledge of its pathology is practically a failure. While much has been written upon the pyorrhoeic stage of interstitial gingivitis and its treatment, during the past two decades, no new principle has been advanced w^hereby the parts can be restored to a healthy condition, or whereby the disease can be prevented. The disease is admittedly on the increase. This seems at first sight to indicate that dental prophylaxis and treatment, so far as this disease is concerned, are failures. Nearly three decades ago I felt and expressed the necessity for more extended study (Dental Cosmos, 1886, page 689) of the clinical aspects and etiology of this disease. Even daring apparently diverse and separated studies, such as those related to dental and maxillary irregularities and degeneracy, the ne- cessity for this has forced itself still further upon me. In the present study, the disease has, of necessity, been considered from the broad standpoint of general pathology. In all in- stances where possible personal elements of error are present, these have been eliminated by having researches made by more than one observer. The attempt has been made to summarize all researches on the subject. So much have opinions been intermingled that it is possible that proper credit for priority has unintentionally not been given. The author is under obligation to the following scientists for their kind assistance: Dr. Ludwig Hektoen, Pathologist, Rush Medical College; Dr. Jerome H. Salisbury, Chemist, Rush Medical College; Dr. W. A. Evans, Pathologist, Columbus Med- ical Laboratory, Professor of Pathology, Chicago College of Physicians and Surgeons and Milwaukee Medical College; Dr. J. A. Wesener, Chemist, Columbus Medical Laboratory, Profes-